Robert W Malone, about ADE

4) The reports of equivalent (or perhaps higher in vax recip?) levels of virus in vaccinated and unvaccinated is odd.
5) Viral infection/replication is necessary but not sufficient for disease - disease is the patient hyperinflamm response

— Robert W Malone, MD (@RWMaloneMD) August 1, 2021

6) The clinical trials were not designed to detect ADE, despite it being a major risk for corona vax development
7) FDA specifically acknowledged that ADE was a risk, and suggested focused trials were warranted - but did not require them.
8) I find no trials to rule in/out ADE

— Robert W Malone, MD (@RWMaloneMD) August 1, 2021

And finally, ADE has many mechanisms. The easiest to explain and understand is facilitation of monocyte infection. So that is the example I use. The mechanism of ADE observed in prior coronavirus vax efforts is not very clear.

— Robert W Malone, MD (@RWMaloneMD) August 1, 2021